Issue link: http://cp.revolio.com/i/924515
Page 34| Abby's Magazine - www.AbbysHealthAndNutrition.com extra energy as fat for future needs when food may not be available. The fact that insulin lowers your blood sugar is merely a "side effect" of this energy storage process. Taking more insulin just makes you fa er! Your body's cells become desensi zed to insulin, lep n, and other hormones, by being overexposed to these hormones— be it by ea ng food that causes excessive secre on, or by injec on. Diabetes treatments that concentrate merely on lowering blood sugar by adding insulin therefore tend to worsen rather than remedy the actual problem of metabolic miscommunica on. As Dr. Rosedale has previously stated: "Type 2 diabetes is brought on by constantly having too much insulin and lep n circula ng secondary to the same diet that has been recommended to treat diabetes and heart disease, a high carbohydrate, low-fat diet. Then giving these diabe cs more insulin is adding gasoline to the fire. Doctors couldn't be doing more harm if they tried." Leptin—An Oft-Ignored KEY Player in Type 2 Diabetes Development While much conven onal advice centers around insulin, lep n is another hormone that plays an integral role in the development of type 2 diabetes. Lep n is produced in your fat and other cells, and one of its primary roles is regula ng your appe te and body weight. Lep n tells your brain when to eat, how much to eat, and most importantly, when to stop ea ng. Lep n also instructs your brain as to what to do with the available energy. Now remember, when your blood sugar becomes elevated, insulin is released to direct the extra energy into storage—the majority of which is stored as fat, and lep n is produced in these fat cells. The more fat you have, the more lep n is produced. Furthermore, as the sugar gets metabolized in your fat cells, the fat releases further surges in lep n. This is why I typically talk about insulin and lep n resistance, as they work in tandem. Moreover, lep n is largely responsible for the accuracy of insulin signaling and whether or not you become insulin-resistant. If you're insulin resistant, you're more than likely lep n resistant as well, especially if you're overweight or obese. Why? Because when you develop lep n resistance, your brain can no longer hear lep n's signals, resul ng in chronic hunger, overea ng, inability to properly burn fat and, typically, obesity. Insulin resistance, and ul mately type 2 diabetes, follow suit. Just as with insulin, the only known way to reestablish proper lep n signaling is through proper diet. High consump on of carbohydrates, especially fructose, is again the prime culprit and the root cause of lep n resistance. Lack of exercise and abnormal gut flora also contribute and/or exacerbate insulin and lep n resistance. Lep n's importance in blood glucose control and diabetes is powerfully illustrated by recent studies that show its ability, even in low doses, to lower blood glucose in both type 1 and 2 diabe cs, and this is an exci ng new poten al treatment. New Warning: Insulin Can Rapidly Produce Type 1 Diabetes in Type 2 Diabetics Please understand that medica ons and supplements are not the answer for type 2 diabetes. Diabetes drugs fail to address the underlying problem, and many, like Avandia,